Alzheimer Immunotherapy Trial Grounded: Time to Reassess Safety and Vaccine Design
injecting Aβ1-42 more than two years ago (Schenk et al., 1999), I was very surprised they did it in that
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Neural Stem Cells to the Rescue—Can Neuroreplacement Ever Become a Treatment for Alzheimer's?
subsequent improvement of cognitive function (Qu et al., 2001) reinforces the potential feasibility of HNSC
A "Mitochondrial Cascade Hypothesis" for Sporadic Alzheimer's Disease
(Nunomura et al., 2004) showing that where Aβ exists, there is actually less evidence of oxidative stress in
Making a BioMark on Alzheimer Disease
milestone paper by Pratico et al., 2001 on isoprostanes in AD and also papers by Montine et al., 2005 and
Sports Concussions, Dementia, and APOE Genotyping: What Can Scientists Tell the Public? What’s Up for Research?
the best large population data out there. See Guskiewicz et al., 2005 and Guskiewicz et al., 2007.
ADCS Prevention Instrument Project
appeared in the October-December 2006 supplement. Steve Ferris et al. 0 WEBINAR Monday, March 12, 2007-
Targeting Tumor Necrosis Factor—A Therapeutic Strategy for AD
inhibitor, which blocks its translocation: dehydroxymethylepoxyquinomicin (Kubota et al., 2007). Lovastatin
Mice on Trial? Issues in the Design of Drug Studies
neurological disease is the discovery by Hauser et al., recently published in the New England Journal of
Alzheimer Presenilins in the Nuclear Membrane, Interphase Kinetochores, and Centrosomes Suggest a Role in Chromosome Segregation
IL-1-positive microglia in DS brain of all ages (Griffin et al, 1989). Mqxia: Hi, nice to be back here. June
Who Should Use the New Diagnostic Guidelines? The Debate Continues
covered this important milestone with commentary (see Dubois et al., 2007), in the news (see ARF related
Isolation of Single Immunohistochemically Identified Whole Neuronal Cell Bodies from Post-Mortem Human Brain for Simultaneous Anlaysis of Multiple Gene Expression
et al., (Int. J. Neurosci., 1990, 55:81-88.). Question 3) What percentage of the brains that were
In Search of γ-Secretase
experiment (Wolfe, et al.) are provocative, but they do not conclusively show whether the presenilins are
Pin1, Tau and the Cell Cycle in Alzheimer Disease
mitosis[ref.5]). Summary of Paper Under Discussion In the paper being presently discussed, Lu et al. provide data
Is Alzheimer's an Autoimmune Disease?
neurons that light up. Dara Dickstein We have recently published in Microcirculation (Ujiie et al., 2003)
Gain or Loss of Function—Time to Shake up Assumptions on γ-Secretase in Alzheimer Disease?
Aβ40 levels whereas only some drove up Aβ42. Kumar-Singh et al. also found that loss of Ab40 correlated
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